Motuel bT. If you want to subscribe to this journal, see our rates You can purchase this item in Pay Per View: The owners of this website hereby guarantee to respect the legal confidentiality conditions, applicable in France, and not to disclose this data to third parties. You may thus request that your data, should it be inaccurate, incomplete, unclear, outdated, not be used or stored, be corrected, clarified, updated or deleted. The owners of this website hereby guarantee to respect the legal confidentiality conditions, applicable in France, and not to disclose this data to third parties.
|Published (Last):||27 April 2007|
|PDF File Size:||3.55 Mb|
|ePub File Size:||14.12 Mb|
|Price:||Free* [*Free Regsitration Required]|
This article has been cited by other articles in PMC. Ocular fundoscopy available in 12 patients showed bilateral papilloedema in eight and optic disk atrophy in four. Clinical evolution was particularly noticeable in five patients because of chronic two patients or acute after lumbar shunting or puncture: three patients, one death tonsillar herniation. The remaining 11 had type II fistulas drainage into a sinus, with abnormal retrograde venous drainage into sinuses or cortical veins.
Stenosis or thrombosis of the sinus es distal to the fistula was present in five patients. The cerebral venous drainage was abnormal in all patients. Normal cerebral angiography should be added as a fifth criterion of benign intracranial hypertension. The cerebral venous drainage pattern must be carefully studied by contralateral carotid and vertebral artery injections to correctly evaluate the impairment of the cerebral venous outflow. Lumbar CSF diversion puncture or shunting may induce acute tonsillar herniation and should be avoided absolutely.
DAVF may induce intracranial hypertension, which has a poor long term prognosis and may lead to an important loss of visual acuity and chronic tonsillar herniation. Consequently, patients with intracranial hypertension must be treated, even agressively, to obliterate the fistula or at least to reduce the arterial flow and to restore a normal cerebral venous drainage.
Patients in whom the fistula is not obliterated after an endovascular therapeutic procedure, need continous clinical and angiographical follow up.
Selected References These references are in PubMed. This may not be the complete list of references from this article. Johnston I, Paterson A. Benign intracranial hypertension. Diagnosis and prognosis. CSF pressure and circulation. The pseudotumor syndrome. Disorders of cerebrospinal fluid circulation causing intracranial hypertension without ventriculomegaly. Arch Neurol. Cerebral dural arteriovenous fistulas: clinical and angiographic correlation with a revised classification of venous drainage.
Benign forms of intracranial hypertension; toxic and otitic hydrocephalus. Douze cas. Rev Neurol Paris ; 1 — The incidence of pseudotumor cerebri. Population studies in Iowa and Louisiana. Venous sinus occlusion with a dural arteriovenous malformation of the posterior fossa. Surg Neurol. Pathogenesis of pseudotumor cerebri syndromes. Follow-up study of cases of pseudotumor cerebri.
Acta Neurochir Wien ;18 4 — Pseudotumor cerebri: clinical profile and visual outcome in 63 patients. Mayo Clin Proc. Headache associated with changes in intracranial pressure. Idiopathic intracranial hypertension. A prospective study of 50 patients. Cerebral venous thrombosis. Neurol Clin. Etude clinique, physiopathologique et nosographique. Magnetic resonance imaging in lateral sinus hypoplasia and thrombosis. Etude comparative. Dural arteriovenous malformations and papilledema.
Dural arteriovenous malformations. J Neurosurg. Arteriovenous malformations of the transverse dural sinus. Neurological manifestations of intracranial dural arteriovenous malformations. Clinical syndromes of arteriovenous malformations of the transverse-sigmoid sinus. J Neurol Neurosurg Psychiatry. Fistulae of the lateral sinus. J Neuroradiol. Unusual clinical manifestations of dural arteriovenous malformations. The minor symptoms of increased intracranial pressure: patients with benign intracranial hypertension.
Long-term changes in intracranial dural arteriovenous fistulae leading to worsening in the type of venous drainage. Dural arteriovenous malformation of the major venous sinuses: an acquired lesion.
Intracranial dural arteriovenous malformations. Microvascularization of the intracranial dura mater. Surg Radiol Anat. Two cases of dural arteriovenous malformation occurring after intracranial surgery. A variant of arteriovenous fistulas within the wall of dural sinuses. Results of combined surgical and endovascular therapy.
Etiological evaluation of dural arteriovenous malformations of the lateral and sigmoid sinuses based on histopathological examinations. Dural posterior fossa AVM producing raised sagittal simus pressure. Case report. Long term follow-up of 43 pure dural arteriovenous fistulae AVF of the lateral sinus.
Dural fistulas involving the transverse and sigmoid sinuses: results of treatment in 28 patients. Transvenous embolization of dural fistulas involving the transverse and sigmoid sinuses. Dural arteriovenous fistulas involving the inferior petrosal sinus: angiographic findings in six patients.
Percutaneous transvenous embolization through the thrombosed sinus in transverse sinus dural fistula. Cerebral dural arteriovenous fistulas: percutaneous transvenous embolization.
Hypertension intracrânienne idiopathique
Œdème papillaire de stase bilatéral révélant une hypertension intracrânienne bénigne
Idiopathisch intrakranielle Hypertension (Pseudotumor cerebri) - Inselspital Bern - Neurochirurgie
Hypertension intracrânienne - nature, causes et traitement